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|firstname=Aritri
|firstname=Aritri
|title=Dr.
|title=Dr.
|institution=Department of Biochemistry Β 
|institution=[[File:BirA.jpg|right|200px|Aritri Bir]]
Β 
Department of Biochemistry Β 


Institute of Post-graduate Medical Education & Research IPGMER
Institute of Post-graduate Medical Education & Research IPGMER
|address=244B, A J C Bose Road Β 
|address=244B, A J C Bose Road
Β 
|area code=700020
700020 Kolkata, India
|city=Kolkata
|country=India
}}
}}
{{Labelingperson}}
{{Labelingperson}}
* [http://www.oroboros.at/?IOC73_kalkota IOC73]
==Participated at==
::::* [[MiPNet18.08_MiP2013|MiP2013 Obergurgl AT]]
::::* [[IOC79|IOC79 Obergurgl AT]]
::::* [[IOC73|IOC73 Kolkata IN]]

Latest revision as of 15:15, 13 February 2018

Name Bir Aritri, Dr.
Institution
Aritri Bir

Department of Biochemistry

Institute of Post-graduate Medical Education & Research IPGMER

Address 244B, A J C Bose Road, 700020
City Kolkata
State/Province
Country India
Email [email protected]
Weblink
O2k-Network Lab IN Haldia Chakrabarti S


Labels:



Publications

 PublishedReference
Ganguly 2022 MitoFit2022Ganguly U, Bir A, Chakrabarti S (2022) Cytotoxicity of mitochondrial Complex I inhibitor rotenone: a complex interplay of cell death pathways. https://doi.org/10.26124/mitofit:2022-0013.v2 β€” 2022-11-22 published in Bioenerg Commun 2022.14.
Ganguly 2022 BEC2022Ganguly U, Bir A, Chakrabarti S (2022) Cytotoxicity of mitochondrial Complex I inhibitor rotenone: a complex interplay of cell death pathways. Bioenerg Commun 2022.14. https://doi.org/10.26124/bec:2022-0014

Abstracts

 PublishedReference
Chakrabarti 2022 Abstract Bioblast2022Not presented
Chakrabarti Sasanka
Ganguly U, Bir A, Chakrabarti Sasanka (2022) Cytotoxicity of mitochondrial Complex I inhibitor rotenone: a complex interplay of cell death pathways. Bioblast 2022: BEC Inaugural Conference. In: https://doi.org/10.26124/bec:2022-0001
Β»MitoFit PreprintΒ«
Bir 2013 Abstract MiP20132013Bir A, Sen O, Anand S, Banerjee P, Sahoo A, Khemka V, Cappai R, Chakrabarti S (2013) Ξ±-Synuclein mediated alterations in mitochondrial oxidative phosphorylation system: Implications in the pathogenesis of Parkinson’s disease. Mitochondr Physiol Network 18.08.

Participated at

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